Correction to “Celastrol Ameliorates Neuronal Mitochondrial Dysfunction Induced by Intracerebral Hemorrhage via Targeting cAMP‐Activated Exchange Protein‐1”
Source abstract
Abstract
Adv. Sci. 2024, e2307556. https://doi.org/10.1002/advs.202307556 Figure 5 Enrichment analysis to identify potential target signaling pathway for celastrol on neurons. A) Rbfox3 was used as a marker for neurons. B) 3D-Principal component analysis (PCA) was employed to identify the subclusters with the highest variance. C) The strength of interactions between neuron 8 and other cell types was evaluated through CellChat analysis of intercellular communication. D) The analysis of cell–cell communication was performed using CellChat to demonstrate the quantification of interactions between neuron 8 and other cellular populations. E) The volcano plot illustrates the differential expression of genes between the ICH+Cel group and the ICH+vehicle group. F) A Gene Ontology (GO) enrichment analysis was performed to identify potential signaling pathways regulated by celastrol following ICH, with an average log2 fold change >0.25. G) Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway analysis was conducted to identify potential signaling pathways regulated by celastrol following ICH, with an average log2 fold change >0.25. These changes do not affect the results or conclusions of this study. The authors apologize for any inconvenience caused.
Source: openalex_inverted_index · Quality: baseline
Research context
OpenAlex topics: Natural Compounds in Disease Treatment · Neurological Disease Mechanisms and Treatments
SciVX discovery tags are terminology-based. OpenAlex topics are source-provided classifications.
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