SOSTDC1 Nuclear Translocation Facilitates BTIC Maintenance and CHD1‐Mediated HR Repair to Promote Tumor Progression and Olaparib Resistance in TNBC
Abstract
Research context
OpenAlex topics: PARP inhibition in cancer therapy · DNA Repair Mechanisms · Cell death mechanisms and regulation
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Affiliations
Fudan University Shanghai Cancer Center & Institutes of Biomedical Sciences State Key Laboratory of Genetic Engineering Cancer Institutes Key Laboratory of Breast Cancer in Shanghai The Shanghai Key Laboratory of Medical Epigenetics Shanghai Key Laboratory of Radiation Oncology The International Co‐laboratory of Medical Epigenetics and Metabolism Ministry of Science and Technology Shanghai Medical College Fudan University Shanghai 200032 China
Research Center for Translational Medicine Shanghai East Hospital Tongji University School of Medicine Shanghai 200120 China
Department of Breast Surgery Shanghai Cancer Center and Cancer Institute Fudan University Shanghai 200032 P. R. China
Department of Thyroid Breast and Vascular Surgery Xijing Hospital The Fourth Military Medical University Xi'an 710032 P. R. China
Department of Breast Surgery Fudan University Shanghai Cancer Center Shanghai 200032 China
Key Laboratory of Breast Cancer in Shanghai Department of Oncology Shanghai Medical College Fudan University Shanghai 200032 China
Key Laboratory of Animal Models and Human Disease Mechanisms of Chinese Academy of Sciences and Yunnan Province Kunming Institute of Zoology Kunming 650201 China
Academy of Biomedical Engineering & The Third Affiliated Hospital Kunming Medical University Kunming 650118 China
Jiangsu Key Lab of Cancer Biomarkers Prevention and Treatment Collaborative Innovation Center for Cancer Medicine Nanjing Medical University Nanjing 211166 China
Funding
National Key Research and Development Program of China · National Natural Science Foundation of China · Program of Shanghai Academic Research Leader